Application of Finite Elastic Theory to the Deformation of Rubbery Materials

The purpose of this discussion, then, is to show how the nature of the strain energy function can be deduced from experiments on rubbery materials

Gamma-ray Luminosity and Photon Index Evolution of FSRQ Blazars and Contribution to the Gamma-ray Background

We present the redshift evolutions and distributions of the gamma-ray luminosity and photon spectral index of flat spectrum radio quasar (FSRQ) type blazars, using non-parametric methods to obtain the evolutions and distributions directly from the data. The sample we use for analysis consists of almost all FSRQs observed with a greater than approximately 7 sigma detection threshold in the first year catalog of the Fermi Gamma-ray Space Telescope's Large Area Telescope, with redshfits as determined from optical spectroscopy by Shaw et al. We find that FSQRs undergo rapid gamma-ray luminosity evolution, but negligible photon index evolution, with redshift. With these evolutions accounted for we determine the density evolution and luminosity function of FSRQs, and calculate their total contribution to the extragalactic gamma-ray background radiation, resolved and unresolved, which is found to be 16(+10/-4)%, in agreement with previous studies.Comment: 9 pages, 10 figures, Accepted to Ap

Muon anomalous magnetic moment from effective supersymmetry

We present a detailed analysis on the possible maximal value of the muon (g-2) (= 2 a_mu) within the context of effective SUSY models with R parity conservation. First of all, the mixing among the second and the third family sleptons can contribute at one loop level to the a_mu(SUSY) and tau -> mu gamma simultaneously. One finds that the a_mu(SUSY) can be as large as (10-20)*10^-10 for any tan beta, imposing the upper limit on the tau -> mu gamma branching ratio. Furthermore, the two-loop Barr-Zee type contributions to a_mu(SUSY) can be significant for large tan beta, if a stop is light and mu and A_t are large enough (O(1) TeV). In this case, it is possible to have a_mu(SUSY) upto O(10)*10^-10 without conflicting with tau -> l gamma. We conclude that the possible maximal value for a_mu(SUSY) is about 20*10^-10 for any tan beta. Therefore the BNL experiment on the muon a_mu can exclude the effective SUSY models only if the measured deviation is larger than \sim 30*10^-10.Comment: 10 pages, 3 figure

Contribution of b→sggb \to sgg through the QCD anomaly in exclusive decays B±→(η′,η)(K±,K∗±)B^{\pm}\to (\eta^{\prime},\eta)(K^{\pm}, K^{*\pm}) and B0→(η′,η)(K0,K∗0)B^{0}\to (\eta^{\prime},\eta)(K^{0},K^{*0})

We compute the decay rates for the exclusive decays $B^{\pm} \to (\eta^{\prime},\eta) (K^{\pm}, K^{*\pm})$ and $B^{0}\to (\eta^{\prime},\eta) (K^{0}, K^{*0})$ in a QCD-improved factorization framework by including the contribution from the process $b\to sgg \to s (\eta^{\prime}, \eta)$ through the QCD anomaly. This method provides an alternative estimate of the contribution $b \to s c\bar{c} \to s(\eta,\eta^\prime)$ to these decays as compared to the one using the intrinsic charm content of the $\eta^{\prime}$ and $\eta$ mesons determined through the decays $J/\psi \to (\eta,\eta^\prime ,\eta_c) \gamma$. The resulting branching ratios are compared with the CLEO data on $B^{\pm} \to \eta^{\prime} K^{\pm}$ and $B^{0} \to \eta^{\prime} K^{0}$ and predictions are made for the rest.Comment: 16 pages including 4 postscript figures; uses epsfig. The most recent branching ratios from CLEO, ref. [5], are taken into account. The theory part is unchange

Methylation of CpG island is not a ubiquitous mechanism for the loss of oestrogen receptor in breast cancer cells.

Methylation has been shown to play an important role in the down-regulation of oestrogen receptors (ER) in breast cancer cells. One critical question that remains unclear is whether methylation can account for the loss of ER expression in cells derived from an ER-positive cell line. This laboratory has established an in vitro cell system using long-term growth of human ER-positive breast cancer cell line T47D in oestrogen-free medium. A clonal cell line, T47D:C4:2 (C4:2), has been characterized. Unlike T47D:A18 (A18), which is a T47D line maintained in oestrogen medium, C4:2 has lost the expression of ER and hormone responsiveness. DNA fingerprinting and restriction fragment length polymorphism (RFLP) analysis results confirmed that C4:2 was of the same lineage as A18. These cell lines provide an invaluable system to study the mechanism of ER expression and regulatory pathways leading to hormone-independent growth. The results here clearly demonstrate that the ER CpG island in C4:2 cells remains unmethylated. The loss of ER in the cell line must be due to mechanisms other than methylation. We also evaluated the ER CpG island in the MDA-MB-231:10A (10A) cell line, which is a clone from the MDA-MB-231 line obtained from ATCC and the DNA from the MDA-MB-231 cell line used in the original report. Unlike the cell line from the report, which showed a full methylation pattern in the island, the 10A line only showed a partial methylation pattern in the CpG island. Possible mechanisms pertaining to the heterogeneous methylation pattern of the ER CpG island in the breast cancer cells are discussed